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Erschienen in: Surgical Endoscopy 5/2009

01.05.2009

Clinical relevance of laparoscopically diagnosed hiatal hernia

verfasst von: Yves Van Nieuwenhove, Jeroen Sonck, Boudewijn De Waele, Peter Potvlieghe, Georges Delvaux, Patrick Haentjens

Erschienen in: Surgical Endoscopy | Ausgabe 5/2009

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Abstract

Background

To determine the clinical relevance of a laparoscopically diagnosed hiatal hernia.

Methods

Consecutive patients undergoing an elective laparoscopy were prospectively recruited. We assessed preoperative gastroesophageal reflux symptoms using a validated score, and documented the presence or absence of a hiatal hernia during laparoscopy.

Results

Of the 95 evaluable patients, 42 (44%) had a hiatal hernia. The mean age was 49.8 years. Logistic regression analysis indicated that three features were significantly and independently associated with hiatal hernia: a higher reflux score (odds ratio [OR] 2.44; 95% confidence interval [CI] 1.48-4.05; p < 0.001), low body mass index (BMI) (OR 0.83; 95% CI 0.70–0.98; p = 0.029), and type of surgery (OR 0.34; 95% CI 0.14–0.92; p = 0.033). The diagnostic accuracy of a reflux score of more than 2 was 81%, with a sensitivity, specificity, positive predictive value, and negative predictive value of 76%, 85%, 80%, and 82%, respectively. The likelihood ratio for a positive result was 5.05.

Conclusion

Hiatal hernia is common in this population of surgical patients undergoing an elective laparoscopy. Patients with reflux symptoms or a low BMI were more likely to have a hiatal hernia. With a reflux score of more than 2, the probability of finding a hiatal hernia during laparoscopy is 80%.
Literatur
1.
Zurück zum Zitat Locke GR III, Talley NJ, Fett SL, Zinsmeister AR, Melton LJ III (1997) Prevalence and clinical spectrum of gastroesophageal reflux: a population-based study in Olmsted County, Minnesota. Gastroenterology 112:1448–1456PubMedCrossRef Locke GR III, Talley NJ, Fett SL, Zinsmeister AR, Melton LJ III (1997) Prevalence and clinical spectrum of gastroesophageal reflux: a population-based study in Olmsted County, Minnesota. Gastroenterology 112:1448–1456PubMedCrossRef
2.
Zurück zum Zitat Nebel OT, Fornes MF, Castell DO (1976) Symptomatic gastroesophageal reflux: incidence and precipitating factors. Am J Dig Dis 21:953–956PubMedCrossRef Nebel OT, Fornes MF, Castell DO (1976) Symptomatic gastroesophageal reflux: incidence and precipitating factors. Am J Dig Dis 21:953–956PubMedCrossRef
3.
Zurück zum Zitat Ronkainen J, Aro P, Storskrubb T, Johansson SE, Lind T, Bolling-Sternevald E, Graffner H, Vieth M, Stolte M, Engstrand L, Talley NJ, Agreus L (2005) High prevalence of gastroesophageal reflux symptoms and esophagitis with or without symptoms in the general adult Swedish population: a Kalixanda study report. Scand J Gastroenterol 40:275–285PubMedCrossRef Ronkainen J, Aro P, Storskrubb T, Johansson SE, Lind T, Bolling-Sternevald E, Graffner H, Vieth M, Stolte M, Engstrand L, Talley NJ, Agreus L (2005) High prevalence of gastroesophageal reflux symptoms and esophagitis with or without symptoms in the general adult Swedish population: a Kalixanda study report. Scand J Gastroenterol 40:275–285PubMedCrossRef
4.
Zurück zum Zitat Cohen S, Harris LD (1971) Does hiatus hernia affect competence of the gastroesophageal sphincter? N Engl J Med 284:1053–1056PubMed Cohen S, Harris LD (1971) Does hiatus hernia affect competence of the gastroesophageal sphincter? N Engl J Med 284:1053–1056PubMed
5.
Zurück zum Zitat Dodds WJ, Dent J, Hogan WJ, Helm JF, Hauser R, Patel GK, Egide MS (1982) Mechanisms of gastroesophageal reflux in patients with reflux esophagitis. N Engl J Med 307:1547–1552PubMed Dodds WJ, Dent J, Hogan WJ, Helm JF, Hauser R, Patel GK, Egide MS (1982) Mechanisms of gastroesophageal reflux in patients with reflux esophagitis. N Engl J Med 307:1547–1552PubMed
6.
Zurück zum Zitat Kahrilas PJ, Shi G, Manka M, Joehl RJ (2000) Increased frequency of transient lower esophageal sphincter relaxation induced by gastric distention in reflux patients with hiatal hernia. Gastroenterology 118:688–695PubMedCrossRef Kahrilas PJ, Shi G, Manka M, Joehl RJ (2000) Increased frequency of transient lower esophageal sphincter relaxation induced by gastric distention in reflux patients with hiatal hernia. Gastroenterology 118:688–695PubMedCrossRef
7.
Zurück zum Zitat van Herwaarden MA, Samsom M, Smout AJ (2000) Excess gastroesophageal reflux in patients with hiatus hernia is caused by mechanisms other than transient LES relaxations. Gastroenterology 119:1439–1446PubMedCrossRef van Herwaarden MA, Samsom M, Smout AJ (2000) Excess gastroesophageal reflux in patients with hiatus hernia is caused by mechanisms other than transient LES relaxations. Gastroenterology 119:1439–1446PubMedCrossRef
8.
Zurück zum Zitat Panzuto F, Di Giulio E, Capurso G, Baccini F, D’Ambra G, Delle Fave G, Annibale B (2004) Large hiatal hernia in patients with iron deficiency anaemia: a prospective study on prevalence and treatment. Aliment Pharmacol Ther 19:663–670PubMedCrossRef Panzuto F, Di Giulio E, Capurso G, Baccini F, D’Ambra G, Delle Fave G, Annibale B (2004) Large hiatal hernia in patients with iron deficiency anaemia: a prospective study on prevalence and treatment. Aliment Pharmacol Ther 19:663–670PubMedCrossRef
9.
Zurück zum Zitat Allen CJ, Parameswaran K, Belda J, Anvari M (2000) Reproducibility, validity, and responsiveness of a disease-specific symptom questionnaire for gastroesophageal reflux disease. Dis Esophagus 13:265–270PubMedCrossRef Allen CJ, Parameswaran K, Belda J, Anvari M (2000) Reproducibility, validity, and responsiveness of a disease-specific symptom questionnaire for gastroesophageal reflux disease. Dis Esophagus 13:265–270PubMedCrossRef
10.
Zurück zum Zitat Gordon C, Kang JY, Neild PJ, Maxwell JD (2004) The role of the hiatus hernia in gastro-oesophageal reflux disease. Aliment Pharmacol Ther 20:719–732PubMedCrossRef Gordon C, Kang JY, Neild PJ, Maxwell JD (2004) The role of the hiatus hernia in gastro-oesophageal reflux disease. Aliment Pharmacol Ther 20:719–732PubMedCrossRef
11.
Zurück zum Zitat Bombeck CT, Dillard DH, Nyhus LM (1966) Muscular anatomy of the gastroesophageal junction and role of phrenoesophageal ligament; autopsy study of sphincter mechanism. Ann Surg 164:643–654PubMedCrossRef Bombeck CT, Dillard DH, Nyhus LM (1966) Muscular anatomy of the gastroesophageal junction and role of phrenoesophageal ligament; autopsy study of sphincter mechanism. Ann Surg 164:643–654PubMedCrossRef
12.
Zurück zum Zitat Sloan S, Rademaker AW, Kahrilas PJ (1992) Determinants of gastroesophageal junction incompetence: hiatal hernia, lower esophageal sphincter, or both? Ann Intern Med 117:977–982PubMed Sloan S, Rademaker AW, Kahrilas PJ (1992) Determinants of gastroesophageal junction incompetence: hiatal hernia, lower esophageal sphincter, or both? Ann Intern Med 117:977–982PubMed
13.
Zurück zum Zitat Corley DA, Kubo A (2006) Body mass index and gastroesophageal reflux disease: a systematic review and meta-analysis. Am J Gastroenterol 101:2619–2628PubMedCrossRef Corley DA, Kubo A (2006) Body mass index and gastroesophageal reflux disease: a systematic review and meta-analysis. Am J Gastroenterol 101:2619–2628PubMedCrossRef
14.
Zurück zum Zitat Hampel H, Abraham NS, El-Serag HB (2005) Meta-analysis: obesity and the risk for gastroesophageal reflux disease and its complications. Ann Intern Med 143:199–211PubMed Hampel H, Abraham NS, El-Serag HB (2005) Meta-analysis: obesity and the risk for gastroesophageal reflux disease and its complications. Ann Intern Med 143:199–211PubMed
15.
Zurück zum Zitat El-Serag HB, Johanson SF (2002) Risk factors for the severity of erosive esophagitis in Helicobacter pylori-negative patients with gastroesophageal reflux disease. Scand J Gastroenterol 37:899–904PubMedCrossRef El-Serag HB, Johanson SF (2002) Risk factors for the severity of erosive esophagitis in Helicobacter pylori-negative patients with gastroesophageal reflux disease. Scand J Gastroenterol 37:899–904PubMedCrossRef
16.
Zurück zum Zitat Stene-Larsen G, Weberg R, Froyshov Larsen I, Bjortuft O, Hoel B, Berstad A (1988) Relationship of overweight to hiatus hernia and reflux oesophagitis. Scand J Gastroenterol 23:427–432PubMedCrossRef Stene-Larsen G, Weberg R, Froyshov Larsen I, Bjortuft O, Hoel B, Berstad A (1988) Relationship of overweight to hiatus hernia and reflux oesophagitis. Scand J Gastroenterol 23:427–432PubMedCrossRef
17.
Zurück zum Zitat Wilson LJ, Ma W, Hirschowitz BI (1999) Association of obesity with hiatal hernia and esophagitis. Am J Gastroenterol 94:2840–2844PubMedCrossRef Wilson LJ, Ma W, Hirschowitz BI (1999) Association of obesity with hiatal hernia and esophagitis. Am J Gastroenterol 94:2840–2844PubMedCrossRef
18.
Zurück zum Zitat Wu AH, Tseng CC, Bernstein L (2003) Hiatal hernia, reflux symptoms, body size, and risk of esophageal and gastric adenocarcinoma. Cancer 98:940–948PubMedCrossRef Wu AH, Tseng CC, Bernstein L (2003) Hiatal hernia, reflux symptoms, body size, and risk of esophageal and gastric adenocarcinoma. Cancer 98:940–948PubMedCrossRef
19.
Zurück zum Zitat Ovrebo KK, Hatlebakk JG, Viste A, Bassoe HH, Svanes K (1998) Gastroesophageal reflux in morbidly obese patients treated with gastric banding or vertical banded gastroplasty. Ann Surg 228:51–58PubMedCrossRef Ovrebo KK, Hatlebakk JG, Viste A, Bassoe HH, Svanes K (1998) Gastroesophageal reflux in morbidly obese patients treated with gastric banding or vertical banded gastroplasty. Ann Surg 228:51–58PubMedCrossRef
20.
Zurück zum Zitat Capron JP, Payenneville H, Dumont M, Dupas JL, Lorriaux A (1978) Evidence for an association between cholelithiasis and hiatus hernia. Lancet 2:329–331PubMed Capron JP, Payenneville H, Dumont M, Dupas JL, Lorriaux A (1978) Evidence for an association between cholelithiasis and hiatus hernia. Lancet 2:329–331PubMed
Metadaten
Titel
Clinical relevance of laparoscopically diagnosed hiatal hernia
verfasst von
Yves Van Nieuwenhove
Jeroen Sonck
Boudewijn De Waele
Peter Potvlieghe
Georges Delvaux
Patrick Haentjens
Publikationsdatum
01.05.2009
Verlag
Springer-Verlag
Erschienen in
Surgical Endoscopy / Ausgabe 5/2009
Print ISSN: 0930-2794
Elektronische ISSN: 1432-2218
DOI
https://doi.org/10.1007/s00464-008-9970-4

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