CCS is one of the well-known complications of continuous curvilinear capsulorrhexis. It has been described as an exaggerated fibrotic response that can lead to a reduction in the size of the anterior capsulotomy [
1,
2]. Thereafter, CCS results in impaired visual function secondary to the opacity in pupillary area. It has been associated with uveitis, pseudoexfoliation, myotonic dystrophy, and retinitis pigmentosa [
3]. CCS has also occurred with small capsulorrhexis openings of less than 6 mm diameter, and Acrylic IOL revealed lowest rates [
4]. The present case was an unusual, compared with previously reported CCS cases that were treated with Nd:YAG laser radial capsulotomy, because an uneventful surgery without zonular weakness was performed, the patient had no underlying disease except for a history of an angle-closure attack; furthermore, anterior capsulorhexis opening was completely occluded. In addition, proliferation of fibrotic tissue was relatively prominent in this case, rather than the appearance of capsular phimosis. In previous studies, CCS was thought to have been caused by two underlying mechanisms; shrinkage of the capsulorhexis leading to the formation of smaller diameter capsular opening, and the development of a fibrocellular membrane caused by lens epithelial cells closing remaining central opening [
5]. Spang et al. noted that complete occluded anterior capsulorhexis opening was filled with proliferated lens epithelial cells under light microscopy, and the cellular elements revealed a positive reaction for actin filament upon immunohistochemical analysis [
5]. In our opinion, this is an uncommon case showing a total occlusion of the anterior capsulorhexis opening with prominent fibrotic proliferation pattern after an uneventful cataract surgery. Additionally, the occlusion could be removed with a non-invasive procedure, and was maintained clearly for a couple of years.