Skip to main content
Erschienen in: Clinical Autonomic Research 2/2022

22.03.2022 | Editorial

Mechanisms of post-prandial symptoms in postural tachycardia syndrome and other updates on recent autonomic research

verfasst von: Mitchell G. Miglis, Nicholas Larsen, Srikanth Muppidi

Erschienen in: Clinical Autonomic Research | Ausgabe 2/2022

Einloggen, um Zugang zu erhalten

Excerpt

Most patients with postural tachycardia syndrome (POTS) report worsening of symptoms after meals rich in carbohydrates, however, the precise underlying mechanism is unknown. In their recent study, “Worsening postural tachycardia syndrome is associated with increased glucose-dependent insulinotropic polypeptide secretion,” Breier and colleagues [ 1] evaluated the secretion of glucose-dependent insulinotropic polypeptide (GIP), a splanchnic vasodilator, to investigate the pathophysiological mechanisms involved in post-prandial symptom exacerbation in POTS. …
Literatur
7.
Zurück zum Zitat Courand PY, Lantelme P (2017) Stretching the carotid sinus to treat resistant hypertension. Lancet 390:2610–2612 CrossRefPubMed Courand PY, Lantelme P (2017) Stretching the carotid sinus to treat resistant hypertension. Lancet 390:2610–2612 CrossRefPubMed
Metadaten
Titel
Mechanisms of post-prandial symptoms in postural tachycardia syndrome and other updates on recent autonomic research
verfasst von
Mitchell G. Miglis
Nicholas Larsen
Srikanth Muppidi
Publikationsdatum
22.03.2022
Verlag
Springer Berlin Heidelberg
Erschienen in
Clinical Autonomic Research / Ausgabe 2/2022
Print ISSN: 0959-9851
Elektronische ISSN: 1619-1560
DOI
https://doi.org/10.1007/s10286-022-00863-4

Weitere Artikel der Ausgabe 2/2022

Clinical Autonomic Research 2/2022 Zur Ausgabe