Clinical studyElectrophysiologic characteristics of concealed bypass tracts: Clinical and electrocardiographic correlates☆
References (26)
- et al.
Concealed bypass of the atrioventricular node in patients with paroxysmal supraventricular tachycardias revealed by intracardiac stimulation and Verapamil
Am. J Cardiol
(1974) - et al.
Clinical and electrophysiologic observations in patients with concealed accessory atrioventricular bypass tracts
Am J Cardiol
(1977) Concealed anomalous cardiac conduction pathways: a frequent cause of supraventricular tachycardia
Am J Cardiol
(1977)- et al.
Mode of initiation of reciprocating tachycardia during programmed ventricular stimulation in the Wolff-Parkinson-White syndrome
Am J Cardiol
(1977) - et al.
Direct study of left atrial P waves
Am J Cardiol
(1967) - et al.
Demonstration of dual atrioventricular nodal pathways in man
Am J Cardiol
(1974) - et al.
Mechanisms of atrioventricular junctional tachycardia
Am J Cardiol
(1977) - et al.
Antegrade block in accessory pathways with retrograde conduction in reciprocating tachycardia
Eur J Cardiol
(1975) - et al.
Analysis of re-entry mechanisms in three patients with concealed Wolff-Parkinson-White syndrome
Circulation
(1975) - et al.
Les syndrome de Wolff Parkinson White de type A inapparents ou latents en rhythm sinusal
Arch Mal Coeur Vaiss
(1973)
The role of an accessory atrio-ventricular pathway in reciprocal tachycardia: observations in patients with and without the Wolff-Parkinson-White syndrome
Circulation
Wolff-Parkinson-White syndrome. The problem, evaluation and surgical correction
Circulation
Reciprocating tachycardia in overt and latent pre-excitation. Influence of functional bundle branch block on the rate of the tachycardia
Eur J Cardiol
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Shorter post pacing interval during initiation of regular narrow QRS tachycardia. What is the mechanism?
2021, Journal of ElectrocardiologyCitation Excerpt :Another possible explanation is presence of dual AV nodal pathways, with only the first tachycardia beat utilizing the fast AV nodal pathway. The reported incidence of dual AV nodal pathways in patients with accessory pathways has been estimated to be 40% [4]. The accessory pathway is mapped at 3 O clock of the mitral annulus and successfully ablated.
Ventricular-triggered atrial pacing: A new maneuver for slow-fast atrioventricular nodal reentrant tachycardia
2020, Heart RhythmCitation Excerpt :This finding however lacks in sensitivity as, at baseline, only approximately 60% of patients with AVNRT exhibit this property.5 More importantly, this finding also lacks in specificity for the diagnosis of AVNRT as such an anterograde “jump” can be demonstrated in approximately 40% of patients with AVRT due to concealed accessory pathways6 and up to 70% of patients with idiopathic ventricular tachycardia.2 From a theoretical standpoint, longitudinal dissociation or sudden conduction delay across relatively refractory tissue within a single pathway may also produce a discontinuous curve,7,8 and hence this finding alone cannot be considered complete proof of the presence of 2 anatomically or functionally distinct pathways between which reentry can occur.
Everybody has Brugada syndrome until proven otherwise?
2015, Heart RhythmVentricular Preexcitation (Wolff-Parkinson-White Syndrome and Its Variants)
2008, Chou's Electrocardiography in Clinical Practice: Adult and PediatricAtrioventricular Junctional Rhythms
2008, Chou's Electrocardiography in Clinical Practice: Adult and PediatricAtrioventricular nodal reentrant tachycardia associated with idiopathic ventricular tachycardia: clinical and electrophysiologic characteristics
2007, Journal of ElectrocardiologyCitation Excerpt :Anterograde and/or retrograde dual AVN pathways have been observed in 3% to 25% of patients without known SVT during electrophysiologic study; however, patients generally do not have AVN echo beats and therefore lack a functional retrograde fast pathway.19,30,31 Dual AVN physiology has also been demonstrated in 8% to 35% of patients with Wolff-Parkinson-White syndrome.17,32,33 In this study, we found a similar 27% incidence of dual AVN physiology in the subset of patients with VT and structural heart disease.
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This work was supported in part by grants from the American Heart Association, Southeastern Pennsylvania Chapter, Philadelphia, Pennsylvania and by Grant HL 14807 from the National Institutes of Health, Bethesda, Maryland.
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Recipient of Career Development Investigatorship, American Heart Association, Southeastern Pennsylvania Chapter.