Left ventricular size, mass and function in relation to the duration and quantity of heavy drinking in alcoholics☆
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2018, AACE Clinical Case ReportsPrognostic Impact and Predictors of Ejection Fraction Recovery in Patients With Alcoholic Cardiomyopathy
2018, Revista Espanola de CardiologiaQuantifying the contribution of alcohol to cardiomyopathy: A systematic review
2017, AlcoholCitation Excerpt :They found a higher proportion of cases drinking more than the recommended weekly intake (three standard drinks per day – 30 g for men and two standard drinks/day – 20 g for women; 40% vs. 24%), a higher proportion of cases screening positive in the CAGE (27% vs. 16%), and higher lifetime consumption (31,200 vs. 7904 standard drinks). The importance of level of alcohol consumption is additionally supported by a population study, finding positive correlations between amount of drinking and size or mass of the heart (Manolio, Levy, Garrison, Castelli, & Kannel, 1991), as well as by clinical studies correlating lifetime alcohol exposure with parameters of cardiac impairment (Fernández-Solà et al., 2000; Kino, Imamitchi, Morigutchi, Kawamura, & Takatsu, 1981; Lazarević et al., 2000; but see Kupari, Koskinen, & Suokas, 1991). The fact that it takes heavy drinking with levels of 80 g of pure alcohol per day and more was also corroborated by two studies with other methodology.
The relationship of alcohol consumption with left ventricular mass in people 35 years old or older in rural areas of Western China
2017, Journal of the American Society of HypertensionLeft ventricular strain and rotation by 2-d speckle tracking echocardiography identify early alcoholic cardiomyopathy
2016, Ultrasound in Medicine and BiologyNatural history and prognostic factors in alcoholic cardiomyopathy
2015, JACC: Heart FailureCitation Excerpt :Finally, our study did not find differences in clinical outcomes between ACM patients who abstain completely from alcohol and those who reduce intake to a moderate degree. Excessive and prolonged alcohol intake leads to systolic dysfunction in some alcohol abusers (15–19), most likely due to a genetic susceptibility (20). Given the high incidence of alcohol intake in industrialized nations, alcohol has been proposed as the major contributor to nonischemic DCM in Western countries (8,9,11).
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This study was supported in part by the Finnish Foundation for Alcohol Research, by the Yrjö Jahnsson Foundation and by the Paavo Nurmi Foundation.