Alteration of renal function in hyperthyroidism: Increased tubular secretion of creatinine and decreased distal tubule delivery of chloride
References (19)
- et al.
The thyroid and the kidney
Kidney Int
(1974) - et al.
Creatinine: An inadequate filtration marker in glomerular disease
Am J Med
(1980) - et al.
Evidence for a prostaglandin-independent defect in chloride reabsorption in the loop of Henle as a proximal cause of Bartter's syndrome
Am J Med
(1978) - et al.
Effect of triiodothyronine on the synthesis and degradation of renal cortical (Na+K+)-adenosine triphosphatase
J Biol Chem
(1976) - et al.
An elevation of BUN/creatinine ratio in patients with hyperthyroidism
Horm Metab Res
(1986) - et al.
Plasma natriuretic peptide, plasma renin activity and aldosterone during treatment of hyperthyroidism due to Graves' disease
Horm Metab Res
(1989) - et al.
Renal function and electrolyte levels in hyperthyroidism: Urinary protein excretion and the plasma concentrations of urea, creatinine, uric acid, hydrogen ion and electrolytes
Clin Endocrinol
(1989) A hypothesis for the thyroid hormone-induced increase in RPF and GFR
Nephron
(1977)- et al.
Kidney function in various thyroid states
J Clin Endocrinol Metab
(1961)
Cited by (38)
Assisting the diagnosis of Graves' hyperthyroidism with pattern recognition methods and a set of three routine tests parameters, and their correlations with free T4 levels: Extension to male patients
2011, Biomedicine and PharmacotherapyCitation Excerpt :When we tried different combinations of parameters, the use of γ-GTP, and AST, ALT often erroneously placed patients with hepatic disease into the hyperthyroid zone, whereas ALP, S-Cr, and TC gave the best screening results as was found for female cases. As for the set of two parameters (ALP and TC) recommended in the guidelines for Graves’ disease [21,22] both the classification ability of learning samples and the screening ability of the test samples were worse than the results for the set of three parameters, which indicates the importance of S-Cre [23]. Fig. 2 shows a SOM that was constructed using the three best routine tests (ALP, S-Cr, and TC) as training samples, and 133 test samples (black or white) from patients who had undergone routine tests at the Department of Comprehensive Medicine of Tohoku University Hospital after complaining of illness, have been projected onto it.
Cystatin C as a Biomarker in Kidney Disease
2011, Biomarkers of Kidney DiseaseInteractions between thyroid and kidney function in pathological conditions of these organ systems: A review
2009, General and Comparative EndocrinologyCitation Excerpt :The intrarenal feedback mechanism increases the GFR to cope with the threatening hypoperfusion and the escape of urine entering the distal tubule, which has to be replaced by the delivery of proximal tubule fluid (Straub, 1977). Thyroid hormones increase mRNA expression of chloride channels (ClC) in a dose-dependent way (Shirota et al., 1992; Santos et al., 2003) and also increase the activity of ClC and Cl absorption in the proximal tubule and Henle’s loop. Tubulo-glomerular feedback adjusts GFR after a decreased chloride load is sensed within the distal tubule by the macula densa (Graves et al., 1994).
Hyperthyroidism and the kidney
2006, Clinical Techniques in Small Animal Practice