Flow cytometric studies on actin polymerization in PMN cells from chronic myeloid leukemia (CML) patients
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Preclinical model for identification of therapeutic targets for CML offers clues for handling imatinib resistance
2015, Biomedicine and PharmacotherapyCitation Excerpt :Moreover, actin-binding domain of bcr-abl is required for full transforming activity. In CML PMNL, stimulation of actin polymerization and various actin dependent functions are defective [15–18]. Therefore, actin was selected as the second parameter.
Differences in the microtubule organization between normal and CML granulocytes after stimulation with chemotactic peptide
2000, Cell Biology InternationalWhole-blood incubation method to study neutrophil cytoskeletal dynamics
1997, Journal of Immunological MethodsChronic myeloid leukemia granulocytes have lower amounts of cytoplasmic actin
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