Journal of the Autonomic Nervous System
Research paperSympathetic outflow to muscles during vasovagal syncope
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Clinical features of prolonged tilt-induced hypotension with an apparent vasovagal mechanism, but without syncope
2019, Autonomic Neuroscience: Basic and ClinicalThe pathophysiology of the vasovagal response
2018, Heart RhythmCitation Excerpt :These extraordinary scientific developments enabled clinicians and researchers to study noninvasively the beat-to-beat hemodynamics of laboratory-induced vasovagal syncope. Over the same period, impedance measurements have demonstrated directional changes in segmental blood volume during tilt and microneurography has allowed us to monitor efferent muscle vasoconstrictor sympathetic activity (MSNA).3,4 As a result of this approach, researchers have been able to “sequence” the hemodynamic changes during orthostasis in much greater detail, and so relationships between variables (eg, mean arterial pressure [MAP] and CO) have become clearer.
Predictive value of very low frequency at spectral analysis among patients with unexplained syncope assessed by head-up tilt testing
2018, Archives of Cardiovascular DiseasesCitation Excerpt :Firstly, vasovagal syncope could be caused by acute systemic vasodilation, induced by strong contractions of an empty left ventricle – the so called “empty heart syndrome” [20]. Secondly, it has been proposed that the main driver of vasovagal syncope is the loss of sympathetic tone, causing relaxation of vascular smooth muscle [4,21]. Moreover, according to several microneurographic studies, a loss of sympathetic activity is not always a prerequisite for (pre)syncope [22–24].
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