CHAPTER 28 - Nutrition of Laboratory Mice
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Effects of high fat diets on rodent liver bioenergetics and oxidative imbalance
2016, Redox BiologyCitation Excerpt :Due to the lack of information in some papers, a few assumptions were made to facilitate comparisons: Most authors refer to fat content as percentage of energy from fat, since ad libitum feeding will be determined by energy requirements [59,60]. However, if the description was made by fat weight and the metabolized energy per weight of food was available, energy from fat was calculated by assuming (as defined by FAO) that 1 g of fat=9 kcal=37 kJ (FAO, 2013).
Administration of caseinomacropeptide-enriched extract to mice enhances the calcium content of femur in a low-calcium diet
2015, International Dairy JournalCitation Excerpt :Then, the aforementioned studies allow determination of Ca content in the femur during short term (3 weeks) administration of CMP. This feeding period is also coincident with the period of full development in mice that goes from weaning (3 weeks of age) to adulthood (6 weeks of age) (Ritskes-Hoitinga, 2004). No significant differences in the body weight of animals were observed (Fig. 4) during the 3-week feeding period, nor after 8 weeks of oral administration of the product under study (data not shown).
Bile acid-regulated peroxisome proliferator-activated receptor-α (PPARα) activity underlies circadian expression of intestinal peptide absorption transporter PepT1/Slc15a1
2014, Journal of Biological ChemistryCitation Excerpt :We first investigated the relationship between temporal feeding patterns and the accumulation of fatty and bile acids in intestinal epithelial cells. As reported previously (25), mice consumed ∼80% of their food during the dark phase when they were fed ad libitum (Fig. 1A). Under this feeding condition, the amount of free fatty acids in the intestinal epithelial cells of mice exhibited an obvious circadian oscillation with higher levels being observed during the dark phase (Fig. 1B).
Selenium in the redox regulation of the Nrf2 and the Wnt pathway
2013, Methods in EnzymologyCitation Excerpt :With this background in mind, a study was designed to mimic the impact of a moderate selenium deficiency, as it might be expected from common variations in nutritional habits (Kipp et al., 2009). Four week-old mice were fed a diet containing 0.086 mg Se/kg for 6 weeks, which is about half the recommended daily intake for mice (0.150 mg/kg diet) (Ritskes-Hoitinga, 2004). For control, selenium was added in the form of selenomethionine up to the recommended dosage.
Methods for long-term 17β-estradiol administration to mice
2012, General and Comparative EndocrinologyLoss of GPx2 increases apoptosis, mitosis, and GPx1 expression in the intestine of mice
2010, Free Radical Biology and MedicineCitation Excerpt :Apoptosis and mitosis were analyzed at 20 weeks of age. For adjusting the selenium status, the Se-adequate and Se-supplemented diets were obtained by mixing selenomethionine (Acros, Geel, Belgium) into a torula-yeast-based selenium-poor diet (No. C1045, with 50% carbohydrate, 17% protein, 5% fat, 4% fiber, and a mixture of micronutrients; Altromin, Lage, Germany) containing 0.086 mg Se/kg [17] to yield a selenium content of 0.15 mg/kg for the selenium-adequate diet, corresponding to the dietary reference intake for mice [18], and a selenium content of 0.66 mg/kg for the selenium-supplemented diet. All diets were fed as pellets starting directly after weaning.