THE COMMON MIGRAINE ATTACK MAY NOT BE INITIATED BY CEREBRAL ISCHAEMIA
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Cited by (137)
Migraine with brainstem aura
2024, Handbook of Clinical NeurologyA new era for migraine: The role of calcitonin gene-related peptide in the trigeminovascular system
2020, Progress in Brain ResearchCitation Excerpt :In 1944, Aristides Leão described cortical spreading depression (Leão, 1944). Olesen et al. found that regional cerebral blood flow decreased during migraine aura, followed by hyperperfusion during headache, and finally, a return to normal blood flow after the headache subsides (Olesen et al., 1981). In the 1980s, the neurogenic inflammation theory was proposed based on the observation that proinflammatory mediators are released, along with vasodilation during migraine attacks (McCulloch et al., 1986; Moskowitz and Cutrer, 1993).
Ambient air pollution exposure and risk of migraine: Synergistic effect with high temperature
2018, Environment InternationalCitation Excerpt :This might be another reason for the diminished, non-significant association between air pollution and migraine on high-temperature days when we defined high-temperature days using a 99th percentile temperature (28.7 °C) (Fig. 4). Previous studies have found that reduced regional cerebral blood flow is prominent in patients with aura, but not in patients without aura (Olesen et al., 1981a; Olesen et al., 1981b). A clinical study has found different effectiveness of pharmacotherapy according to subtype of migraine (with and without aura) (Hansen et al., 2015).
The role of CGRP in the pathophysiology of migraine and efficacy of CGRP receptor antagonists as acute antimigraine drugs
2009, Pharmacology and TherapeuticsChapter 17 Laser evoked potentials in primary headaches: a possible clinical or research tool?
2006, Supplements to Clinical NeurophysiologyNeuroimaging in the pre-ictal or premonitory phase of migraine: a narrative review
2023, Journal of Headache and Pain