Antibiotics, skin and soft tissue infection and meticillin-resistant Staphylococcus aureus: cause and effect
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2018, Journal of OrthopaedicsCitation Excerpt :Recent epidemiologic and microbiologic research found that in France, two thirds of Staphylococcus aureus are sensitive to Augmentin (amoxicillin and clavulanic acid). On the contrary, the trend is the inverse in the United States [19,20], where two thirds of Staphylococcus aureus are resistant to Augmentin. These microbiological data allow for the administration of empirical antibiotic therapy, while definitive microbiologic results are awaited.
Novel 3-O-carbamoyl erythromycin A derivatives (carbamolides) with activity against resistant staphylococcal and streptococcal isolates
2013, Bioorganic and Medicinal Chemistry LettersControlling hospital MRSA
2013, Journal of Global Antimicrobial ResistanceCitation Excerpt :It is safe to conclude that without antibiotics there would be no MRSA and that the antimicrobial era has been primarily responsible for driving the rapid evolution of S. aureus since the second world war, in a truly epic battle of survival of the fittest. Even in community- and livestock-associated MRSA we see the influence of antimicrobial selection pressure [7]. Transmission of MRSA in the hospital requires a source, which is usually patients in the endemic setting and rarely staff.
Comparison of necrotizing fasciitis and sepsis caused by Vibrio vulnificus and Staphylococcus aureus
2011, Journal of Bone and Joint SurgeryCitation Excerpt :In the past decade, Staphylococcus aureus was reported to be the most common cultured pathogen4,6,17,18. The Panton-Valentine leukocidin (PVL) toxin and several clones carrying different staphylococcal cassette chromosome (SCC) mecA gene cassette types have been reported in association with Staphylococcus aureus skin and soft-tissue infections23–26. MRSA has been reported to have a prevalence of 59% to 75% in soft-tissue infections and 29% to 39% in necrotizing fasciitis1,2,5,8,16–19,27.