Short communicationReduction in open field activity in the absence of memory deficits in the AppNL−G−F knock-in mouse model of Alzheimer’s disease
Section snippets
Funding
LSW was awarded an Australian Rotary Health/Rotary Club of Adelaide Funding Partner Scholarship and an Australian Government Research Training Program Scholarship. Funding was also provided by the Lysosomal Diseases Research Unit, Nutrition and Metabolism Theme, SAHMRI. Funding bodies did not influence design or analysis of this study.
Acknowledgments
We thank Dr Stuart Howell for statistical advice, Leanne Hein for assistance with genotyping, Amanda Wilson for caring for the mice and Yi Ng for Slide Scanner operation.
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