Letter to the editorThymic stromal lymphopoietin and IL-33 promote skin inflammation and vaccinia virus replication in a mouse model of atopic dermatitis
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Cited by (25)
Ginsenoside Rg3 attenuates skin disorders via down-regulation of MDM2/HIF1α signaling pathway
2021, Journal of Ginseng ResearchCitation Excerpt :Serum and skin lesions of patients with atopic dermatitis contained high levels of TSLP [42]. TSLP receptor deficiency led to decreased skin inflammation in mice [43]. TSLP deficiency resulted in reduced skin inflammation in mice [13].
Chloroquine attenuates thymic stromal lymphopoietin production via suppressing caspase-1 signaling in mast cells
2021, Biomedicine and Pharmacotherapyp-coumaric acid, an active ingredient of Panax ginseng, ameliolates atopic dermatitis-like skin lesions through inhibition of thymic stromal lymphopoietin in mice
2021, Journal of Ginseng ResearchCitation Excerpt :Our preliminary experiment showed that exposure to PMACI increased the mRNA expression and production of TSLP in HMC-1 cells [12]. Deficiency of TSLP ameliorated skin inflammation in a murine AD model [34]. Intradermal injection of recombinant TSLP increased scratching behavior in a murine AD model [35].
Report from the National Institute of Allergy and Infectious Diseases workshop on “Atopic dermatitis and the atopic march: Mechanisms and interventions”
2019, Journal of Allergy and Clinical ImmunologyCitation Excerpt :The mechanisms by which these alarmins contribute to allergen sensitization in the skin and initiate cross-talk between the skin, gut, and airway are the subject of intense investigation.128 Mouse models that simulate skin inflammation and AD have been particularly useful for investigating TSLP, IL-33, and IL-25 interactions and identifying target populations and downstream signaling events.128-130 Dr Steve Ziegler and colleagues tested the functional role of IL-33 and the interplay between IL-33 and TSLP in a model of intradermal administration of ovalbumin (OVA) with either TSLP or IL-33, followed by oral allergen challenge.131,132
Staphylococcus aureus Epicutaneous Exposure Drives Skin Inflammation via IL-36-Mediated T Cell Responses
2017, Cell Host and Microbe
Supported by National Institutes of Health grant HHSN272201000020C (to R.S.G.). M.K.O. was funded by the Food Allergy Research & Education, Inc, the HOPE APFED/ARTrust Pilot Grant, the William F. Milton Fund, and the Boston Children's Hospital Pediatric Associates Award.
Disclosure of potential conflict of interest: M. K. Oyoshi receives research funding form the William F. Milton Fund, HOPE APFED/ARTrust Pilot Grant, and Boston Children's Hospital Pediatric Associate Award and has grants pending with Food Allergy Research and Education. R. S. Geha receives research funding from the National Institute of Allergy and Infectious Diseases; serves on the editorial board of the Journal of Allergy and Clinical Immunology; and has patents with Stratagene and receives royalties form Garland Publishing. N. Venturelli declares that he has no relevant conflicts of interest.