Deletion Mapping of Chromosome 1 in Early Onset and Late Onset Breast Tumors – AComparative Study in Eastern India
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2018, Biochemical and Biophysical Research CommunicationsCitation Excerpt :AT-rich interaction domain 1a (ARID1A) is one subunit within the SWItch/Sucrose Non-Fermentable (SWI/SNF) chromatin remodeling complex [14]. It is located in chromosomal region 1p36, which is a region that is often deleted within many different forms of cancer [14,15]. A high frequency of mutation of ARID1A in cancers, specifically endometrioid cancer of the uterus and ovarian clear cell and endometrioid cancer, has revealed the potential of ARID1A to be defined as a tumor suppressor [16,17].
Frequent inactivation of MCC/CTNNBIP1 and overexpression of phospho-beta-catenin<sup>Y654</sup> are associated with breast carcinoma: Clinical and prognostic significance
2016, Biochimica et Biophysica Acta - Molecular Basis of DiseaseRole of the EphB2 receptor in autophagy, apoptosis and invasion in human breast cancer cells
2014, Experimental Cell ResearchCitation Excerpt :For example, deletions at chr. 1p36 region, where EphB2 is localized, were very frequently found in breast cancers [11,62], but the specific role of EphB2 in these cancers is not known. In the present work we assessed EphB2 expression in different models of breast cell lines and human clinical tissues and we identified an important role for this receptor in regulating three different homeostatic processes: autophagy, apoptosis and invasion.
Mitochondria-mediated apoptosis in patients with adenocarcinoma of the breast: Correlation with histological grade and menopausal status
2011, BreastCitation Excerpt :This may be because premenopausal breast tumours tend to be generally more aggressive than breast tumours that develop in women after menopause. This aspect is especially significant in countries like India where the incidence of breast tumours is more common in premenopausal women.32,57 The observations from the present study confirm that the differential expression patterns of Bcl-2 family proteins and caspases are involved in evasion of apoptosis and in the progression of breast cancer.