There were trial–time interactions (
p < 0.001). Cross-sectional antral areas increased in the 2 °C and 37 °C trials compared with the 60 °C trial immediately after consuming water (i.e., post) (10.0 ± 5.0 cm
2 vs. 9.3 ± 1.2 cm
2 vs. 6.8 ± 2.1 cm
2 for the 2 °C, 37 °C and 60 °C trials, respectively; 2 °C vs. 60 °C:
p = 0.030, 95% CI 0.379–7.802, 37 °C vs. 60 °C:
p = 0.0019, 95% CI 0.407–4.556) (Fig.
3). The frequency of gastric contractions differed significantly among the trials (main effect of trial,
p < 0.001, and trial–time interaction:
p < 0.001). Post hoc analyses indicated differences in the frequency of the gastric contractions between trials immediately after (i.e., post) and at 10, 20, 30, 40, 50, and 60 min after consuming water—the frequency of the gastric contractions was lower in the 2 °C trial than the 60 °C trial (post:
p < 0.001, 95% CI 2.818–5.932, 10 min:
p = 0.001, 95% CI 1.202–3.548, 20 min:
p = 0.004, 95% CI 1.158–4.592, 30 min:
p = 0.002, 95% CI 1.333–4.167, 40 min:
p = 0.016, 95% CI 0.512–4.238, 50 min:
p = 0.026, 95% CI 0.341–4.909, 60 min:
p = 0.002, 95% CI 1.608–5.142) (Fig.
4). At immediately after (i.e., post), and at 30, 50, and 60 min after consuming water, the frequency of the gastric contractions was lower in the 37 °C trial than the 60 °C trial (post:
p = 0.011, 95% CI 0.605–3.895, 30 min:
p = 0.034, 95% CI 0.104–2.371, 50 min:
p = 0.024, 95% CI 0.369–4.656, 60 min:
p = 0.005, 95% CI 1.192–5.408). At immediately after (i.e., post) and at 10 min after consuming water, the frequency of the gastric contractions was lower in the 2 °C trial than the 37 °C trial (post:
p = 0.003, 95% CI 0.880–3.370, 10 min:
p < 0.001, 95% CI 1.453–2.872).