Abstract
Acute renal failure is characterized by the sudden onset of azotemia in a previously stable patient, usually in association with oliguria or anuria. It is a common complication of many forms of serious illness and metabolic stress. Not infrequently, it is the accidental consequence of medical intervention(s) [1]. The breakdown of the etiology of acute renal failure in prerenal, renal and postrenal causes is not only conceptually attractive, but often dictates initial therapy as well. Knowledge of the normal regulatory mechanisms of electrolyte and fluid turnover, and the changes therein that accompany antecedent chronic renal insufficiency, is essential in this approach. In the following chapter, we will discuss regulatory mechanisms of fluid and electrolyte turnover as it relates to acute renal failure. For extensive review of normal renal physiology, we refer the readers to well-known reference books [2].
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Gans, R.O.B., Ter Wee, P.M. (1998). Regulatory mechanisms of fluid and electrolyte turnover. In: Critical Care Nephrology. Springer, Dordrecht. https://doi.org/10.1007/978-94-011-5482-6_15
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DOI: https://doi.org/10.1007/978-94-011-5482-6_15
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