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Bacterial Translocation During Traumatic Shock in Baboons

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Pathophysiology of Shock, Sepsis, and Organ Failure

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Abstract

Bacterial translocation secondary to gut damage caused by traumatic shock is a source of posttraumatic sepsis. Enteral bacterial translocation has been implicated as a possible cause of the subsequent development of organ failure. In several series, only one third of multiorgan failure patients with clinical manifestations of sepsis had an identifiable focus of infection (Goris et al. 1985). The majority of intensive care patients show the picture of clinical sepsis without a focus. In these patients, the gut frequently plays an important part as a shock organ involved in the development of sepsis.

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Authors and Affiliations

  1. Ludwig Boltzmann Institute for Experimental and Clinical Traumatology, A-1200, Vienna, Austria

    G. Schlag & H. Redl

  2. Roodeplaat Research Laboratories, Pretoria, South Africa

    J. Davies, C. J. J. van Vuuren & P. Smuts

Authors
  1. G. Schlag
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  2. H. Redl
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  3. J. Davies
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  4. C. J. J. van Vuuren
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  5. P. Smuts
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Editors and Affiliations

  1. Ludwig-Boltzmann Institute for Experimental and Clinical Traumatology, Donaueschingenstraße 13, A-1200, Vienna, Austria

    Günther Schlag M. D.  & Heinz Redl Ph. D.  & 

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© 1993 Springer-Verlag Berlin Heidelberg

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Schlag, G., Redl, H., Davies, J., van Vuuren, C.J.J., Smuts, P. (1993). Bacterial Translocation During Traumatic Shock in Baboons. In: Schlag, G., Redl, H. (eds) Pathophysiology of Shock, Sepsis, and Organ Failure. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-76736-4_21

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  • DOI: https://doi.org/10.1007/978-3-642-76736-4_21

  • Publisher Name: Springer, Berlin, Heidelberg

  • Print ISBN: 978-3-642-76738-8

  • Online ISBN: 978-3-642-76736-4

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