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Erschienen in: Journal of Gastroenterology 6/2018

16.04.2018 | Letter to the Editor

Basophils activated via TLR signaling may contribute to pathophysiology of type I autoimmune pancreatitis”

verfasst von: Dimitri Poddighe, Ilaria Brambilla, Gian Luigi Marseglia

Erschienen in: Journal of Gastroenterology | Ausgabe 6/2018

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Excerpt

We read with a great interest the study published by Yaganawa et al. [1] investigating the contribution of basophils in the pathophysiology of type I autoimmune pancreatitis (AIP). Although its pathogenesis is still largely unclear, several data supported an important Th2-driven immune component in this disease where the most remarkable immune-pathological findings are represented by elevated serum levels of IgG4 and tissue infiltration with IgG4-expressing cells [2]. …
Literatur
1.
Zurück zum Zitat Yanagawa M, Uchida K, Ando Y, et al. Basophils activated via TLR signaling may contribute to pathophysiology of type 1 autoimmune pancreatitis. J Gastroenterol. 2018;53(3):449–60.CrossRefPubMed Yanagawa M, Uchida K, Ando Y, et al. Basophils activated via TLR signaling may contribute to pathophysiology of type 1 autoimmune pancreatitis. J Gastroenterol. 2018;53(3):449–60.CrossRefPubMed
2.
Zurück zum Zitat Islam AD, Selmi C, Datta-Mitra A, et al. The changing faces of IgG4-related disease: clinical manifestations and pathogenesis. Autoimmun Rev. 2015;14(10):914–22.CrossRefPubMed Islam AD, Selmi C, Datta-Mitra A, et al. The changing faces of IgG4-related disease: clinical manifestations and pathogenesis. Autoimmun Rev. 2015;14(10):914–22.CrossRefPubMed
3.
Zurück zum Zitat Yamashina M, Nishio A, Nakayama S, et al. Comparative study on experimental autoimmune pancreatitis and its extrapancreatic involvement in mice. Pancreas. 2012;41(8):1255–62.CrossRefPubMed Yamashina M, Nishio A, Nakayama S, et al. Comparative study on experimental autoimmune pancreatitis and its extrapancreatic involvement in mice. Pancreas. 2012;41(8):1255–62.CrossRefPubMed
4.
Zurück zum Zitat Watanabe T, Yamashita K, Sakurai T, et al. Toll-like receptor activation in basophils contributes to the development of IgG4-related disease. J Gastroenterol. 2013;48(2):247–53.CrossRefPubMed Watanabe T, Yamashita K, Sakurai T, et al. Toll-like receptor activation in basophils contributes to the development of IgG4-related disease. J Gastroenterol. 2013;48(2):247–53.CrossRefPubMed
5.
Zurück zum Zitat Karasuyama H, Yamanishi Y. Basophils have emerged as a key player in immunity. Curr Opin Immunol. 2014;31:1–7.CrossRefPubMed Karasuyama H, Yamanishi Y. Basophils have emerged as a key player in immunity. Curr Opin Immunol. 2014;31:1–7.CrossRefPubMed
6.
Zurück zum Zitat Poddighe D, Mathias CB, Freyschmidt EJ, et al. Basophils are rapidly mobilized following initial aeroallergen encounter in naïve mice and provide a priming source of IL-4 in adaptive immune responses. J Biol Regul Homeost Agents. 2014;28(1):91–103.PubMed Poddighe D, Mathias CB, Freyschmidt EJ, et al. Basophils are rapidly mobilized following initial aeroallergen encounter in naïve mice and provide a priming source of IL-4 in adaptive immune responses. J Biol Regul Homeost Agents. 2014;28(1):91–103.PubMed
8.
Zurück zum Zitat Sanjuan MA, Sagar D, Kolbeck R. Role of IgE in autoimmunity. J Allergy Clin Immunol. 2016;137(6):1651–61.CrossRefPubMed Sanjuan MA, Sagar D, Kolbeck R. Role of IgE in autoimmunity. J Allergy Clin Immunol. 2016;137(6):1651–61.CrossRefPubMed
9.
Zurück zum Zitat Pan Q, Gong L, Xiao H, et al. Basophil activation-dependent autoantibody and interleukin-17 production exacerbate systemic lupus erythematosus. Front Immunol. 2017;8:348.PubMedPubMedCentral Pan Q, Gong L, Xiao H, et al. Basophil activation-dependent autoantibody and interleukin-17 production exacerbate systemic lupus erythematosus. Front Immunol. 2017;8:348.PubMedPubMedCentral
10.
Zurück zum Zitat Poddighe D, Marseglia GL. Commentary: basophil activation-dependent autoantibody and interleukin-17 production exacerbate systemic lupus erythematosus. Front Immunol. 2017;8:787.CrossRefPubMedPubMedCentral Poddighe D, Marseglia GL. Commentary: basophil activation-dependent autoantibody and interleukin-17 production exacerbate systemic lupus erythematosus. Front Immunol. 2017;8:787.CrossRefPubMedPubMedCentral
11.
Zurück zum Zitat Poddighe D, Marseglia GL. Is there any relationship between extra-pulmonary manifestations of mycoplasma pneumoniae infection and atopy/respiratory allergy in children? Pediatr Rep. 2016;8(1):6395.CrossRefPubMedPubMedCentral Poddighe D, Marseglia GL. Is there any relationship between extra-pulmonary manifestations of mycoplasma pneumoniae infection and atopy/respiratory allergy in children? Pediatr Rep. 2016;8(1):6395.CrossRefPubMedPubMedCentral
12.
Zurück zum Zitat Valdés Lacasa T, Duarte Borges MA, García Marín A, et al. Acute pancreatitis caused by Mycoplasma pneumoniae: an unusual etiology. Clin J Gastroenterol. 2017;10(3):279–82.CrossRefPubMed Valdés Lacasa T, Duarte Borges MA, García Marín A, et al. Acute pancreatitis caused by Mycoplasma pneumoniae: an unusual etiology. Clin J Gastroenterol. 2017;10(3):279–82.CrossRefPubMed
Metadaten
Titel
Basophils activated via TLR signaling may contribute to pathophysiology of type I autoimmune pancreatitis”
verfasst von
Dimitri Poddighe
Ilaria Brambilla
Gian Luigi Marseglia
Publikationsdatum
16.04.2018
Verlag
Springer Japan
Erschienen in
Journal of Gastroenterology / Ausgabe 6/2018
Print ISSN: 0944-1174
Elektronische ISSN: 1435-5922
DOI
https://doi.org/10.1007/s00535-018-1456-0

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