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Erschienen in: Knee Surgery, Sports Traumatology, Arthroscopy 11/2018

29.01.2018 | Knee

Increases in tibial force imbalance but not changes in tibiofemoral laxities are caused by varus–valgus malalignment of the femoral component in kinematically aligned TKA

verfasst von: Jeremy Riley, Joshua D. Roth, Stephen M. Howell, Maury L. Hull

Erschienen in: Knee Surgery, Sports Traumatology, Arthroscopy | Ausgabe 11/2018

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Abstract

Purpose

The purposes of this study were to quantify the increase in tibial force imbalance (i.e. magnitude of difference between medial and lateral tibial forces) and changes in laxities caused by 2° and 4° of varus–valgus (V–V) malalignment of the femoral component in kinematically aligned total knee arthroplasty (TKA) and use the results to detemine sensitivities to errors in making the distal femoral resections. Because V–V malalignment would introduce the greatest changes in the alignment of the articular surfaces at 0° flexion, the hypotheses were that the greatest increases in tibial force imbalance would occur at 0° flexion, that primarily V–V laxity would significantly change at this flexion angle, and that the tibial force imbalance would increase and laxities would change in proportion to the degree of V–V malalignment.

Methods

Kinematically aligned TKA was performed on ten human cadaveric knee specimens using disposable manual instruments without soft tissue release. One 3D-printed reference femoral component, with unmodified geometry, was aligned to restore the native distal and posterior femoral joint lines. Four 3D-printed femoral components, with modified geometry, introduced V–V malalignments of 2° and 4° from the reference component. Medial and lateral tibial forces were measured during passive knee flexion–extension between 0° to 120° using a custom tibial force sensor. Eight laxities were measured from 0° to 120° flexion using a six degree-of-freedom load application system.

Results

With the tibial component kinematically aligned, the increase in the tibial force imbalance from that of the reference component at 0° of flexion was sensitive to the degree of V–V malalignment of the femoral component. Sensitivities were 54 N/deg (medial tibial force increasing > lateral tibial force) (p < 0.0024) and 44 N/deg (lateral tibial force increasing > medial tibial force) (p < 0.0077) for varus and valgus malalignments, respectively. Varus–valgus malalignment did not significantly change varus, internal–external rotation, anterior–posterior, and compression–distraction laxities from 0° to 120° flexion. At only 30° of flexion, 4° of varus malalignment increased valgus laxity 1° (p = 0.0014).

Conclusion

At 0° flexion, V–V malalignment of the femoral component caused the tibial force imbalance to increase significantly, whereas the laxities were relatively unaffected. Because tibial force imbalance has the potential to adversely affect patient-reported outcomes and satisfaction, surgeons should strive to limit errors in resecting the distal femoral condyles to within ± 0.5 mm which in turn limits the average increase in tibial force imbalance to 68 N. Because laxities were generally unaffected, instability resulting from large increases in laxity is not a clinical concern within the ± 4° range tested.

Level of evidence

Therapeutic, Level II.
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Zurück zum Zitat Whiteside LA, Kasselt MR, Haynes DW (1987) Varus–valgus and rotational stability in rotationally unconstrained total knee arthroplasty. Clin Orthop Relat Res 219:147–157 Whiteside LA, Kasselt MR, Haynes DW (1987) Varus–valgus and rotational stability in rotationally unconstrained total knee arthroplasty. Clin Orthop Relat Res 219:147–157
Metadaten
Titel
Increases in tibial force imbalance but not changes in tibiofemoral laxities are caused by varus–valgus malalignment of the femoral component in kinematically aligned TKA
verfasst von
Jeremy Riley
Joshua D. Roth
Stephen M. Howell
Maury L. Hull
Publikationsdatum
29.01.2018
Verlag
Springer Berlin Heidelberg
Erschienen in
Knee Surgery, Sports Traumatology, Arthroscopy / Ausgabe 11/2018
Print ISSN: 0942-2056
Elektronische ISSN: 1433-7347
DOI
https://doi.org/10.1007/s00167-018-4841-6

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